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Clinical nutrition sel

RDNs are Crucial in Acute Care Settings

Registered Dietitian Nutritionists (RDNs) who work in acute care settings are crucial for ensuring patients receives proper nutrition interventions and care. When a patient is hospitalized, an RDN has a certain timeframe after patient admission (96 hours in my hospital) in which they must review the patient’s chart. While charting, the RDN must learn why the patient was admitted, what their current status is, what their blood serum laboratory results are, what medications they are on that may negatively impact electrolytes or certain nutrients (FNMI), go through their anthropometric measurements and look for any significant weight changes – all of which is literally putting pieces together to get a picture of the patient before seeing the patient in their room – the RDN is also assessing if the patient may be at risk of, or suffering from, malnutrition. One of the favorite parts of my clinical rotation was putting puzzle pieces together while reviewing the patient’s chart yet going up to see the patient to put the final puzzle pieces together. Conducting Nutrition Focused Physical Exams (NFPE) will allow the dietitian to see if the patient has lost fat and muscle stores on their body. This is important when screening for malnutrition and also adds “more pieces to the puzzle.” When an RDN interviews a patient, they are gathering background – how is the patient’s appetite while in the hospital, how much of their meals have they been consuming, have they experienced any nausea, vomiting, constipation or diarrhea, are they experiencing chewing or swallowing difficulties, do they think they’ve lost an abnormal amount of weight within a short amount of time, etc. As a dietetic intern, speaking to patients was the absolute favorite part of my clinical rotation. It is in speaking to the patients that we not only humanize them, but we are able to conduct NFPEs and put the final nutrition puzzle pieces together. During my time as a dietetic intern, I was able to identify patients who needed a referral for SLP (my RD preceptor put in the referral request), I was also able to identify a patient who suffered from moderate protein-calorie malnutrition, and based on my nutrition assessment note, let the medical provider know. After I wrote the patient’s nutrition assessment, my RD preceptor suggested moderate protein-calorie malnutrition be added to the patient’s chart (I could not do this as a dietetic intern) and the medical provider signed in acknowledgement and protein calorie malnutrition was added to the patient’s “problems” lists in his permanent medical record. Once a patient is identified as a malnutrition risk or is suffering from malnutrition, the RDN can ask the patient if they are open and willing to try an oral nutrition supplement (ONS). There are several different types of ONS and the type of ONS the patient receives depends on their medical condition and nutritional status. For example, a patient with several wounds may be given LiquaCel (100 kcal, 16 g PRO) TID or BID to receive extra protein and key micronutrients needed for wound healing. Alternatively, a patient may be given Nepro Carb Steady (420 kcal, 19 g PRO, 38 g CHO) to receive additional calories and protein to help stop unintended weight loss. RDNs ensure weight stabilization occurs and does everything possible to stop unintended weight loss while the patient is hospitalized. Additionally, RDNs are responsible for tube feed calculations and finding a formula that will best fit the patient’s needs. If a patient comes from home on a tube feed, and the hospital does not have the exact formula the patient is fed at home, it is the RDN that finds an acceptable replacement. Additionally, for patients with T2DM, an RDN may identify that bolus feeds may not be the best approach and that perhaps the patient should receive continuous feed to keep blood glucose levels more stable. These are just a few examples of the critical role RDNs play in an acute care setting. The interdisciplinary team and patients alike benefit from the personalized patient nutrition care and monitoring RDNs conduct in an acute care setting.

Reflection

During my clinical rotation, I learned that RDNs play an essential role in the Interdisciplinary team. Many times providers wanted us to give nutrition consults to acute care patients who were overweight. It is not appropriate to give weight loss nutrition education to patients who are hospitalized! Instead, these patients received healthy dietary pattern nutrition education and complimentary materials - but weight loss is reserved for the outpatient RDN. Overall, I learned a great deal of how the clinical environment works. This rotation also confirmed my rotation that acute care is not the reason why I decided to pursue a career in nutrition and dietetics. I decided to pursue a career as an RDN to work with IBD pediatric and adult patients. I am still planning on doing that, and also sports nutrition for fun on the side!

Pharmacology & MNT

Reflection

Lasix (furosemide) is used often in the acute care setting and I saw Lasix use in large percentages of patients daily. During my rotation, there were several patients who needed diuretics due to CHF, HTN, CKD, etc. Many patients suffered from a combination of CHF, CKD State 3a or higher, HTN and, as a result, presented to the ER with severe edema in conjunction with why they were admitted to the hospital (CHF, etc). These patients were often put on furosemide, which is a loop diuretic, to help aid in releasing the fluid their bodies were holding onto. When I was on day 2 of my clinical rotation, I will never forget the time in interdisciplinary rounds when the provider stated that the patient had “lost 13 pounds overnight, so that is a very positive sign.” I almost fell over and couldn’t understand how losing 13-lbs overnight could be a good thing. I asked my Preceptor how a patient losing 13-lbs overnight in an acute care setting (or in any setting) could be a good thing, and she explained to me that the patient lost fluid weight. First, I didn’t realize that a patient could hold on to several pounds of fluid (one patient lost over 34 lbs of fluid over the course of ~3 days). Second, I quickly learned that diuretics such as furosemide can put a patient at risk for hyponatremia, hypokalemia, hypochloremia, and hypomagnesemia. I learned it is crucial to monitor patient’s electrolyte levels when on diuretics. I became very curious about loop diuretics so decided to conduct further research at home and found that Lasix (furosemide) and bumetanide (Bumex) not only deplete electrolytes, but also deplete calcium, thiamine, B6, and vitamin C (depending on the dose).(1,2) Additionally, I learned that thiazide diuretics, such as hydrochlorothiazide, can also lead to hypokalemia, hypomagnesemia, hyponatremia, hypercalcemia and hyperchloremic alkalosis and when used in conjunction with loop diuretics or in doses >25 mg may cause electrolyte derangements.(3) Additionally thiazide diuretics may also lead to metabolic acidosis, hypercalcemia, hyperlipidemia, hyperglycemia and increase the risk of pancreatitis.(3) Lastly, thiazide diuretics should be taken in the morning with breakfast, or food.3 It is important for the RDN to know all of these things and to inform the patient (and remind the interdisciplinary team) that this medication must be taken with food. Equally as important, especially for patients with T2DM, hydrochlorothiazide may also cause hyperglycemia in fasting blood glucose levels. This is important to know, especially when reviewing glucose serum labs from patients that are taken prior to breakfast and in a fasting state.(4) Other diuretics that I saw prescribed quite often are potassium-sparing diuretics such as spironolactone. In these cases, it was important to recognize that the patient may develop hyperkalemia. During my internship it was essential that I recognize when a patient was on diuretics and what possible side effects could happen (such as electrolyte derangements, hyperglycemia, etc) so that I could properly monitor their labs and check for potential electrolyte imbalances. It was important to communicate to the patient that they are on diuretics and usually this would also entail explaining to them why they were also on a CARDIAC/low-sodium diet as oftentimes our patients that were on diuretics were suffering from HF/HTN. Diuretics may cause electrolyte imbalances, and because of this, it was my job as a dietetic intern to monitor electrolyte levels in the patient’s daily comprehensive metabolic panel (CMP) labs to check for possible electrolyte imbalances. Lastly, during my internship, I had the opportunity to explain to HF patients why it was important for them to follow a CARDIAC/low-sodium diet – as we want to decrease the possibility of fluid retention, etc. When patients understand the why behind their prescribed diets (CARDIAC/low-sodium), they tend to be more compliant and accepting. Additionally, we received a nutrition consult to give a patient nutrition education on a low-potassium diet as the patient with CKD Stage 3A was admitted for hyperkalemia and osteomyelitis of L4-5,L1-2 with arachnoiditis and discitis. I had the opportunity to give reduced-potassium diet nutrition education to this patient and explained what foods contained high-potassium and how these high-potassium foods should be replaced with low-potassium foods and gave examples of each. I then left a nutrition education handout containing high and low potassium containing foods for the patient to use as a reference once the patient returned home. The patient seemed quite receptive to nutrition education. This patient was also placed on a reduced-potassium diet, per provider request. Click the tab next to this text to view the nutrition assessment consult I wrote on this patient as well that was placed in his permanent medical record. References 1. Huxel C, Raja A, Ollivierre-Lawrence MD. Loop Diuretics. [Updated 2023 May 22]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2025 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK546656/ 2. The University of Arizona. Drug-Nutrient Interactions. Published April 2010. Accessed August 7, 2025. https://awcim.arizona.edu 3. Patel P, Preuss CV. Thiazide Diuretics. [Updated 2025 Jul 6]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2025 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK532918/ 4. Herman LL, Weber P, Bashir K. Hydrochlorothiazide. [Updated 2023 Nov 12]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2025 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK430766/

Evidenced-Based Literature & MNT

During the duration of my clinical rotation I relied heavily on evidence-based literature. Whether I was giving diabetes nutrition education, nutrition education to patients who suffered from HTN and wanted to learn more about the DASH dietary pattern, a patient who was consistently hyperkalemic and needed to learn how to consume a low-potassium dietary pattern – all of this requires knowledge on the most up-to-date scientific literature. When I gave nutrition education on a specific dietary pattern (i.e. following a 60-75 gm/meal consistent CHO dietary pattern to a patient with T2DM or low-sodium intake for patients with CHF and/or HTN) and explained the why behind needing to follow this dietary pattern (i.e. physiological processes, patient outcomes, etc) the patient was much more open to receiving the nutrition education, took the education more seriously and seemed to be more likely to comply with dietary recommendations. The patient was also more engaged in the nutrition education session in the sense that they were more willing to participate in interactive answer and question or answer when I’d ask, “Show me four carbohydrate items from this exchange list that you can build your plate with at each meal.” Nutrition education was given to a patient with diverticulitis (this patient was excited to learn that she could still eat popcorn as she loved popcorn) and I had many opportunities to give nutrition education to patients who suffered from T2DM and CHF, HTN, CKD and other health ailments. Offering ONS also supports evidence-based literature and when it is explained to the patient why the ONS is being offered and how it will help (i.e. Offering LiquaCel for a patient with wounds who needs extra calories and protein or offering Nepro CarbSteady to a patient with CKD (who may also have T2DM) who needs extra calories but needs a beverage with adjusted potassium, sodium and phosphorus and whose weight we want to stabilize). These patients are usually quite receptive. LiquaCel is notorious for tasting like “bug juice” in our hospital, however, patients with wounds are willing to drink this ONS, despite the taste, once they know it will provide extra calories and protein needed for wound healing. Evidence-based literature is literally used in every aspect of patient care in an acute care setting. I also observed my Preceptor use evidence-based literature to explain to families why tube feeding isn’t’ the best route to go for patients with dementia who have gotten to a point where they forget to eat and are truly suffering from the disease, though many of these families still chose to move forward with tube feeding. Providers are more willing to listen and accept an RDs recommendation when evidence-based literature is used to support the RDs recommendations and guidance. I love evidence-based literature and also know its importance, so as a future RD, I would of course use it in my decision making and nutrition education. However, it was great to observe how it is applied in a real-world clinical setting. From making recommendations to providers and explaining why the recommendation is being made and having the scientific literature to support it all the way to using it to explain to the patient why changing their dietary pattern will benefit their particular health ailment. Overall, scientific literature is absolutely necessary in not only clinical practice in an acute care setting, but in all areas an RD may practice – whether it be outpatient, private, etc. ** Here are a few evidence-based nutrition educations that I gave to patients during my clinical rotation: Diabetes Nutrition Education •Talked about how eating adequate fiber will help reduce postprandial BG levels (I read a few studies on this but ultimately kept it simple for the Veteran in my explanation) •Exercise - Explained that walking after eating increases muscle uptake of glucose and thus improves post prandial BG levels •Explained that 30-minutes of exercise can be broken up into walking for 10-minutes after each meal (10 x 3) to make exercise a more attainable goal and to keep the Veteran from feeling so overwhelmed by the thought of completing 30-minutes of exercise all at once •Explained that consuming a consistent meal pattern of 60-75 gm/meal CHO 3x day (or less for snacks) would help maintain stable BG levels or 3-5 smaller meals if Veteran prefers smaller meals •Explained how to build a balanced plate using the diabetes plate as a visual reference for the Veteran •Explained what a CHO exchange is and how the Veteran can "choose 4 or 5 items off the CHO exchange list to build your meal" •Helped a Veteran visualize and "build" a "diabetes plate" meal so he could visualize what his meals would look like once he returned home. During this session he picked 5 CHO exchanges, lean protein sources, and non-starchy vegetable options. •Emphasized the importance of a balanced dietary pattern Here are some (this is not an all-inclusive list) of evidence-based sources I read to prepare for my nutrition educations on fiber, exercise, dietary pattern, lifestyle in T2DM nutrition education: 1. Xie Y, Gou L, Peng M, Zheng J, Chen L. Effects of soluble fiber supplementation on glycemic control in adults with type 2 diabetes mellitus: A systematic review and meta-analysis of randomized controlled trials. Clin Nutr. 2021;40(4):1800-1810. Doi:10.1016/j.clnu.2020.10.032 2. Giuntini EB, Sardá FAH, de Menezes EW. The Effects of Soluble Dietary Fibers on Glycemic Response: An Overview and Futures Perspectives. Foods. 2022;11(23):3934. Published 2022 Dec 6. Doi:10.3390/foods11233934 3. https://www.sciencedirect.com/science/article/pii/S1756464621001493 4. Hashimoto K, Dora K, Murakami Y, et al. Positive impact of a 10-min walk immediately after glucose intake on postprandial glucose levels. Sci Rep. 2025;15(1):22662. Published 2025 Jul 2. Doi:10.1038/s41598-025-07312-y 5. Way KL, Hackett DA, Baker MK, Johnson NA. The Effect of Regular Exercise on Insulin Sensitivity in Type 2 Diabetes Mellitus: A Systematic Review and Meta-Analysis. Diabetes Metab J. 2016;40(4):253-271. Doi:10.4093/dmj.2016.40.4.253 6. Roberts CK, Little JP, Thyfault JP. Modification of insulin sensitivity and glycemic control by activity and exercise. Med Sci Sports Exerc. 2013;45(10):1868-1877. Doi:10.1249/MSS.0b013e318295cdbb 7. Standards of Care in Diabetes 2025: https://diabetesjournals.org/care/issue/48/Supplement_1 8. Bellini A, Scotto di Palumbo A, Nicolò A, Bazzucchi I, Sacchetti M. Exercise Prescription for Postprandial Glycemic Management. Nutrients. 2024;16(8):1170. Published 2024 Apr 14. doi:10.3390/nu16081170 Hyperkalemia Nutrition Education I also had the opportunity to give a patient nutrition education on low-potassium foods from a nutrition consult request by a provider. The provider wanted the Veteran to receive nutrition education on low-potassium foods. I have attached the nutrition assessment I conducted, and the nutrition education is mentioned in this nutrition assessment. The VA had a nutrition education handout already created for this (we only use VA created nutrition education sheets), but I used the NIH Fact Sheet for Health Professionals to prepare for this nutrition education session. The evidence-based source I referred to for this nutrition education: 1. NIH Potassium Fact Sheets for Health Professionals: https://ods.od.nih.gov/factsheets/Potassium-HealthProfessional/ HTN Nutrition Education I also gave HTN nutrition education and used the DASH diet as the recommendation for the patient with HTN. I provided the Veteran with the HTN handout created by the VA. This is the site I referred to while preparing for the nutrition education session and also used while educating the patient: https://www.nhlbi.nih.gov/education/dash/research

Reflection

Proper nutrition education cannot be given to a patient without the Dietetic Intern (or RDN) using the most up-to-date research and scientific literature. The program director of my MSND program is a nutrition science researcher, sports dietitian and wears many hats in the RDN world. One thing she has INSTILLED in us as her students is the importance of always using scientific literature and evidence-based sources in everything we do as future RDNs. I would never conduct a nutrition education session without reading the most up-to-date research. Luckily, because she has instilled this so much in us, much of the nutrition education I gave I already was familiar with the evidence-based nutrition sources. I also read scientific literature for fun, and always find new and interesting studies as well. Overall, everything I did in my dietetic internship was based on scientific-evidence. I feel incredibly lucky to be at King's College as it is a very science-heavy, evidence-based program that set me up well for my clinical rotation.

Importance of having an RDN in a Substance Abuse Residential Rehabilitation Treatment Program 

While completing my clinical rotation, I had the opportunity to work with Veterans who were enrolled in the Substance Abuse Residential Rehabilitation Treatment Program (SAARTP). I would first see many of these Veterans in the acute care setting and once they were stabilized, they would be transferred over to SAARTP (located in a different wing of the hospital). I immediately saw the importance of this program and am thankful that it is offered to Veterans with substance use disorder (SUD). The RD who is assigned to SAARTP is very passionate about her work and does a great job teaching Veterans how to maintain sustainable, healthy dietary patterns when they return home after inpatient rehabilitation. Nutrition care and education is essential in this Veteran population as many of them come to the program underweight, and once they are stabilized and have detoxed from their addictions (this occurs in the acute care setting), their bodies “wake up” and realize it has been starved from food for weeks or even several months. The RDs role in this program is critical and ensures that these Veterans are taken care of while enrolled in the inpatient program (especially for patients who suffer from T2DM, CKD, HTN). These Veterans, through nutrition education and weekly nutrition classes learn how to eat a sustainable, attainable, healthy dietary pattern and receive the necessary skills on how to do so. These Veterans also have the opportunity to receive one-on-one MNT with an RD while they are enrolled in the program. I had the opportunity to give nutrition education to a few Veterans enrolled in the SAARTP program and also conducted a nutrition assessment. While giving nutrition education, I taught a Veteran with HTN the importance of the DASH dietary pattern and the impact sodium has on blood pressure. I answered all nutrition-related questions he had. This Veteran is an EMT, so he had a great understanding of the pathophysiology of HTN but knew little about nutrition interventions when it came to HTN – which is why he requested and received nutrition education by me! I also conducted a few 1-on-1 T2DM nutrition education sessions with Veterans. It was a great experience to know that I may be playing a direct role in their success of avoiding alcohol and/or drug relapses when they graduate from the program and return home. Due to the importance and magnitude of the SAARTP program, I thought it was crucial to conduct further research to learn more about the program’s success rate and its impact on the Veterans it serves. I would hope the government never chooses to do away with this program, as it is extremely powerful and positively impacts the lives of the Veterans it serves. A recent study of 6,177 Veterans found that VA substance use disorder (SUD) residential treatment programs (such as SAARTP) may reduce all-cause mortality deaths by 66%.(1) This is significant and shows the treatment given and the hard work these Veterans put in while recovering from their alcohol and/or drug addictions result in a significant decrease in all-cause mortality. Additionally, mental health and improvement of SUD symptoms were sustained by the one-year study follow-up period.(1) I am very thankful for the opportunity to directly observe how the SAARTP program is positively impacting Veterans lives. I am also incredibly thankful that I was given the opportunity to give nutrition education to Veterans enrolled in this program. It was also amazing to observe “what happens next” after these Veterans recover from their alcohol/drug detoxes (I would first see these Veterans in the acute care setting while going through the detox process). These Veterans were completely different people once the detox was over and rehabilitation began. It is my hope that the SAARTP program continues to grow and reach more VA medical centers as current data suggests this program significantly decreases death from all-cause mortality within one year of program completion. References 1. Dams GM, Ketchen BR, Burden JL, Smith NB. Effectiveness of residential treatment services for veterans with substance use disorders: A propensity score matching evaluation. Drug Alcohol Depend. 2024;255:111081. doi:10.1016/j.drugalcdep.2024.111081

Reflection

As a Veteran myself, I have seen too many struggle with alcohol and drug addiction. I am part of the OEF/OIF military generation - and a lot of things were seen during that time. Unfortunately, ETOH abuse is prevalent in the military and often times goes under the radar and exacerbates once the Veteran is out of the military. I am glad the VA has programs where these Veterans can get the help they need. Many have seen horrors most can never imagine.

T2DM & HTN Nutrition Education 

During my clinical rotation I was able to give numerous diabetes nutrition education and DASH nutrition education to our patients. I assess I gave more diabetes nutrition education than DASH/low sodium nutrition education to heart failure patients and HTN patients (my main focus in these two patient populations was the DASH dietary pattern). Overall, I emphasized diet in conjunction with healthy lifestyle (i.e. sleep hygiene, exercise). Many times, the providers in acute care wanted my Preceptor and I to give “weight management” nutrition education and teach our patients “how to lose weight,” however, in an acute care setting this is not appropriate. We do not want our patients to lose weight while in acute care. It is not appropriate as RDs to give weight loss nutrition education and encourage weight loss in the acute care setting. However, we can set our patients up for success once they leave the hospital by giving them nutrition education on their health ailment while they are an inpatient yet not bring weight into it at all. Nutrition education is still very impactful without bring weight into the conversation. I was able to give weight management diabetes education for patients in the SAARTP program, as they are in recovery from alcohol and/or drug abuse and are in the program because they are ready to stay free from drugs and/or alcohol and many of these patients want to make health and weight related changes as well. Most patients in SAARTP program gain weight while in the 5-to-8 week recovery program, but this is what we want to see as many of these patients are underweight due to choosing to replace food with alcohol and/or do drugs as eating just wasn’t a priority for many of these patients prior to beginning the program. I gave diabetes nutrition education to patients who suffered from T2DM in the acute care setting and to patients enrolled in the SAARTP program. I also gave low-sodium and DASH dietary pattern nutrition education to patients in the acute care setting and to one patient in the SAARTP program who suffered from HTN. Overall, I was given several opportunities to give patients nutrition education for their health ailments. If the patient was in the acute care setting, I gave diabetes nutrition education teaching the patient how to build a balanced plate while focusing on CHO exchanges to build a balanced 60-75 gm/meal but did not focus on weight management, as this would be inappropriate. However, in the non-acute care setting of the inpatient SAARTP program, I gave diabetes education and also focused on the patient’s weight loss goals. It was a great mix of balance, and I learned so much through both of these experiences! To view the nutrition assessment I wrote after seeing a patient who was receiving double portions during his stay in acute care, click on the button next to this text. I explained to this patient that double servings are not possible, but that I could ensure he receives double vegetable portions and healthy snacks in-between meals instead. The patient was receptive to this, agreed and didn’t protest at all! This was a relief. The provider wanted my Preceptor and I to see this patient because the patient’s BMI put him in the Obesity Class III category. Because weight loss is not an appropriate conversation to have with a patient in the acute care setting I instead focused on doubling this patient’s vegetable servings and removing his diet order of “double portions” (though only my Preceptor could officially remove the double portions in our system – I do not have authority as a Dietetic Intern to officially change diet orders in the system).

Reflection

One of the favorite parts of my clinical rotation was giving nutrition education! I was already very familiar with nutrition interventions for T2DM and HTN because we started learning about evidence-based interventions since our first class in the MSND program. It was exciting to finally be able to present this nutrition education to patients in the hospital setting - whether it be acute care or in-patient rehabilitation program. My MSND program prepared me quite well for this, and I enjoyed being able to interact with patients!

Moderate Protein-Calorie Malnutrition

I was able to conduct a nutrition assessment on a 73 y/o patient who was admitted for SOB 2/2 acute on chronic ischemic/systolic HF. This patient reported that he experienced unintentional weight loss of 35 lbs since November 2024 (when the patient had a stent placed). We received a nutrition consult for this patient and I went into great detail screening him. I looked through his entire weight history, especially his weight history since his stent placement in 11/2024, and did not find any evidence that this patient had lost 35 lbs. I was able to determine that this patient lost 17.3 lbs (8.8%) in the past year, though this is not considered significant weight loss by ASPEN/AND malnutrition criteria. Regardless, I was looking forward to conducting a full NFPE on this patient to see if he suffered from muscle wasting and loss of fat stores. I led the interview with the RD watching and discovered very valuable information. First, I was able to observe the fact that this patient showed positive signs of malnutrition. This patient had subcutaneous fat loss in his orbital fat pads, perioral, chest and bilateral muscle depletion in his temple and shoulder. This is just what I (and the RD) could observe but sadly, the patient declined a full NFPE. I was a little disappointed, but did not show it on my face as this would be unprofessional. I wanted to conduct a full NFPE so I could continue to put the “puzzle pieces” together. Based on my full nutrition assessment, and nutrition-focused physical findings that I could observe (sadly I was not able to observe his scapula, thighs or calf muscles), I was able to find, using the ASPEN/AND malnutrition criteria, that this patient met ≥2 of 6 of the ASPEN/AND malnutrition criteria for Moderate Protein-Calorie Malnutrition and wrote in the Nutrition Assessment that I advise adding Moderate Protein-Calorie Malnutrition to the patient’s progress note. Only the RD can suggest to the provider to add moderate protein-calorie malnutrition to the patient’s diagnosis, but based on my findings, she did write this note to the patient’s provider: Malnutrition Addendum: Your patient has been assessed by a dietitian and found to meet the criteria for moderate protein-calorie malnutrition. If in agreement with this nutrition diagnosis, please add to provider documentation. The provider acknowledged this nutrition diagnosis and moderate protein-calorie malnutrition was added to the patient’s chart. It felt great that my nutrition assessment and malnutrition screening led to the patient’s provider adding this nutrition diagnosis to the patient’s chart and will come up on the patient’s “problem” list in his EMR from here on out.

Reflection

I have suffered from severe protein-calorie malnutrition in the past as a result of a severe Crohn's flare-up. I never had a NFPE and only saw an RD once. Outpatient RDN care was not offered to me. It may have been a breakdown in communication with the hospital, either way, it wasn't right. I am so glad that patient's are being screened for protein-calorie malnutrition and was glad to have the opportunity to do this. Learning about the ASPEN/AND malnutrition screening tool, applying this tool in real-life scenario case studies and then applying it in a clinical setting was an amazing full-circle moment for me (especially since my suggestions of adding moderate protein-calorie malnutrition to the patient's "problem list" was recognized by the hospitalist.

Clinical nutrition prerequisites

Nutrition Focused Physical Exam

Prior to watching the video How to Complete a Nutrition Assessment in the Acute Care Setting + NFPE,(1) I was unsure how to complete a nutrition focused physical exam (NFPE). I am a visual learner, so watching the examination take place in a clinical setting was very helpful for me. Several things stood out to me. I am not yet in my clinical rotation, so this was my first exposure to a NFPE. This may sound silly, but I was reminded of the importance of first asking the patient’s full name and date of birth to ensure we are working with the proper patient.(1) This reminded me of the time my gastroenterologist was handed the wrong chart by his MA, so when he came in to speak with me, he started asking me if I was still seeing a psychiatrist to help me cope with my IBD diagnosis. I looked at him confused because I was not under the care of a psychiatrist. We then locked eyes, and it clicked for him that he was not looking at my chart, but instead looking at the chart of a different patient. He looked at the name on the chart and exclaimed, “This is NOT you!” He angrily walked out of the room to scold his staff. I had never seen my GI doc so angry, but the only reason he was able to identify that he was given the wrong patient chart was because he knew my name due to the ample communication and frequent visits that occurred due to the severity of my disease. However, if I was a new patient, or did not have a strong history with my GI doc, he very well may have assumed that he was given the right chart, which could have unnecessarily complicated things in the future. I had not thought of this in several years (it happened in 2009), but I was reminded of this incident when the video started and the RD asked the patient to state her full name and DOB!(1) It may seem like common sense to do so when entering a patient’s room, however, I am not so sure I would have known to do that if I had not watched the video (I’m sure my clinical preceptor would have instructed me to do so). I particularly liked how the RD explained to the patient why she had a nasogastric (NG) tube, what tube feeding does and why it was important for her to receive formula. Explaining to the patient why they need the formula, what the formula is doing for them from a nutritional perspective, and why they must receive the tube feed is crucial as I’ve been in patient forums where patients stated that they refused enteral formula via tube feed or as a nutritional supplement formula because these formulas were “full of chemicals” and they did not want to put these “chemicals” in their body. Honestly, early on in my IBD journey (I was diagnosed in 2006), I also had many of these beliefs. In 2008, I was in a severe flare and couldn’t eat without experiencing extreme abdominal cramping/pain and having to run to the bathroom several times a day. I decided that I needed to find a nutritional supplement to “give me nutrients” but wanted as little “chemicals” in the formula as possible. I can’t remember how I learned about Peptamen, but I ended up ordering several cases of unflavored Peptamen from a non-medical provider on eBay. I remember that the label specifically stated that the unflavored Peptamen cans I had in my possession was designed for tube feeding and not designed for oral supplementation. I now know that ordering Peptamen from a random stranger from eBay is a huge red flag and that Peptamen should be used under medical supervision. However, at the time, I opted to purchase “unflavored” Peptamen because it lacked the “vanilla flavoring” ingredient that the vanilla Peptamen formula contained. It is an understatement to state that drinking an entire can of unflavored Peptamen every morning was disgusting – but I did it because I was desperate for relief from my IBD symptoms. I would fill up a Starbucks cup with ice and drink my formula daily with a straw. This made consuming unflavored Peptamen more tolerable. Looking back, I can’t believe how desperate I was to go to such extremes for symptom relief. Because of my medical history and knowing how fellow patients think and feel, it is extremely important that an RD explain to the patient what the formula is that the patient is on, why they are on it, and then answer any questions a patient may have on the “chemicals” in the formula. Though the patient in the video did not ask any questions about specific or single formula ingredients and why they are necessary, I know the RD would have answered those questions right away as the first thing she did after asking the patient’s name and DOB was explain what the tube feeding was and why it was necessary.(1) As a future RD and as an IBD patient, I appreciated seeing this as it taught me that this approach would immediately address a patient’s potential fear about formula, or formula “ingredients.” I also learned a great deal observing how the RD worded her questions to the patient to better understand the patient’s usual dietary intake. If I was speaking face-to-face with a patient, I would not have thought to ask questions such as “Compared to a size of a deck of cards, what would your serving look like?” or “Do you finish all of the food on your plate?” or “What type of ice-cream do you consume?”(1) However, I recognize I have zero experience in a clinical setting (other than being a patient), so I assess this is why we are watching this video – to learn! It was also extremely helpful to watch the head-to-toe exam as I had never seen this before and would not know where to start or what to do. It stood out to me when the RD asked, “Are there any areas that you’re having pain that you’d like me to avoid?” I know this sounds silly, but I would not intuitively think to ask this, so I am glad I was able to observe this in the video. While watching the RD complete the head-to-toe exam, I reflect on the fact that I have never received a complete nutrition assessment. However, I completely recognize the importance of a nutrition assessment for IBD patients. I have been hospitalized three times, have been on total parenteral nutrition (TPN) twice for several weeks at a time, and have suffered from malnutrition. Sadly, in 2010, I almost died due to severe malnutrition caused by a severe Crohn’s flare. I now wonder if perhaps I could have avoided TPN and experiencing severe malnutrition if I had just received a nutrition assessment while experiencing a disease flare-up. I will never know, but I know that I can (and will) advocate for IBD patients to undergo nutrition assessments. It was also very helpful to hear how the RD explained everything she would be doing during the head-to-toe exam and the care and humanity she showed to the patient. An example of this is when the RD said, “Are there any areas that you’re having pain that you’d like me to avoid?”(1) I know this sounds silly, but I would not intuitively think to ask this, so I am glad I was able to observe this in the video. Additionally, hearing the RD ask for consent to conduct the exam also stood out to me as again, this is not something I would have intuitively thought to ask. The only thing I would have done differently is, when examining the patient’s shoulders, would have told the patient that I was going to unbuckle her gown to examine her shoulder vice just unbuckling the gown. Likewise, I would have told her that I was now going to button her gown back up after I was done with the examination. I don’t think the RD did anything wrong in the video, I just think letting the patient know that you are going to unbuckle their gown gives them a heads-up and may potentially decrease a trigger in a patient who may have experienced a sexual assault (I may be going too far into the weeds on this, but this is how I would have handled it). Overall, what was most helpful in this video was to see the sequence of events, observe how the RD asked the patient questions, and observe how the RD examines the patient. What also stood out to me was how the RD talked the patient through the entire examination. The line of questioning and explanation by the RD to the patient helped me better understand the entire process. Examples of this include when the patient explained she had a dark line on one of her nails, but in response to that, the RD asked “Are there any recent injuries?”(1) The patient then remembered that she slammed her nail in the door at some point. This exchange between the RD and patient taught me a bit because it taught me to always ask follow-up questions to decide if it’s a nutritional deficiency or an injury, as it is important to identify which it is. This point was further underscored when watching the pediatric NFPE video. RD Jodi Wolff stated, “Always ask yourself, ‘Would my patient have a reason to have this deficiency?’”(2,3) Lastly, it was helpful to observe the RD discuss ideas for nutrition intervention based on the assessment findings.(1) Simple strategies, such as adding butter to toast or mixing shredded cheese with scrambled egg, show how small adjustments can help increase caloric consumption while not overwhelming the patient mentally. Additionally, observing how the RD was able to talk the patient into eating lunch by identifying foods the patient likes was great to observe because I think that if a patient had told me they “weren’t hungry” for lunch I most likely would have not interjected. However, now that I’ve observed this video, I have learned strategies to use if I am given the opportunity to conduct a full NFPE when in my clinical rotation. I learned a great deal from the pediatric NFPE video. RD Wolff mentioned that the pediatric NFPE helps determine nutritional status by uncovering signs of malnutrition, nutrient deficiencies or nutrient toxicities.(2) The term nutrient toxicities stood out to me as its not necessarily something I hear on a regular basis but can be detrimental to both pediatric and adult patients. I didn’t realize there were two types of pediatric NFPE exams, which really makes sense as it’s the comprehensive exam and focused exam (review of specific system based on disease state or specific nutrient concern). I learned that a focused exam may also be appropriate if you hear something in the diet history that may be inadequate and the child may be at risk for that deficiency.(2) I was also shocked to learn that low-fat feeding formula can cause EFA deficiency which may present as thinning, sparse hair.(2) In hindsight this makes sense, however, it is not something I would have intuitively thought of. Additionally, the example photos shown were extremely helpful as I’m a visual learner. I have never seen nor heard of a “flag sign,” so it was helpful to learn that this may occur in a malnourished patient because protein is needed to make pigment in the hair.(2) I used to think that this only impacted adults but now know it could also impact pediatric patients. Overall, it was interesting to observe hypopigmentation in the hair of a pediatric patient as again, I have never seen this before.(2) When discussing the eye examination, it was helpful to learn that the most common eye physical findings found in clinical practice is pale conjunctiva, night blindness, and angular palpebritis.(2) What stood out to me the most when the eyes were discussed is that RD Wolff stated that she has observed bitot spots in patients with intestinal failure and severe malabsorption.(2) I also did not realize that pediatric patients can suffer from taste bud atrophy in vitamin B12 and/or vitamin B6 deficiency.(2) Prior to viewing this video I did realize taste bud atrophy existed. Because I want to work with both adult and pediatric IBD patients, it was interesting to learn that naso-labial dermatitis and vesico-bullous lesions may occur in patients who have zinc deficiency.(2,3) RD Wolff explained that the patient shown in the example photo suffered from zinc deficiency as the patient had high stool output from her ostomy, was on parenteral nutrition and was not receiving adequate zinc supplementation – all which resulted in zinc deficiency and a presentation of both naso-labial dermatitis and vesico-bullous lesions.(2) Additionally, chronic diarrhea also puts patients at risk for zinc deficiency.(2,3) This, in addition to learning about bitot spots in patients with intestinal failure and severe malabsorption, will definitely help me better identify these conditions in IBD pediatric patients in the future. Overall, I learned a great deal from the NFPE videos in both pediatric and adult patients. I recognize that I must conduct an NFPE in-person with real patients, on a consistent basis, to fully grasp how it’s done and to get comfortable with the process. I also recognize that the more practice I get, the better I will be at spotting potential micronutrient deficiencies and malnourishment. Lastly, after watching both the adult and pediatric NFPE videos, I conducted a head-totoe examination on my husband and children. It was helpful for me to go through the process, and I will practice on them several times before starting my clinical rotation this summer. My husband and children are well-nourished, so at least I have a baseline! References 1. Dietitians in Nutrition Support. How to Complete a Nutrition Assessment in the Acute Care Setting + NFPE. YouTube. March 29, 2024. Accessed January 24, 2025. https://www.youtube.com/watch?v=8PgoV6l8MCA 2. Wolff J. Growth Summit 2022: Nutrition Focused Physical Exam Part 1. Abbott Nutrition Health Institute. June 6, 2022. Accessed January 25, 2025. https://www.anhi.org/resources/videos/growth-summit-2022/growth-summit-2022-nutritionfocused-physical-exam-1.html 3. Wolff J. Growth Summit 2022: Nutrition Focused Physical Exam Part 2. Abbott Nutrition Health Institute. June 6, 2022. Accessed January 25, 2025. https://www.anhi.org/resources/videos/growth-summit-2022/growth-summit-2022-nutritionfocused-physical-exam-2.html

Reflection

I am excited that I will be starting my clinical rotation soon and am excited to conduct NFPE on real patients. I am saddened the patient is in the hospital, however, I will do what I can to conduct the NFPE with the utmost respect, dignity, and care. It's amazing how watching a video can prepare a dietetic student for the clinical setting. I am definitely better prepared after watching both the adult and pediatric NFPE videos.

Tube Feeding & Parenteral Nutrition

The videos I chose to watch for this reflection were the Enteral Feed Calculations: Standard Formula, Enteral Nutrition Support: Oncology Case Study, Parenteral Nutrition Calculation: Custom 2-in-1 + Lipid Piggyback, and Parenteral Nutrition Case Study – Crohn’s Disease. Before watching these videos, I was not confident in my enteral and parenteral nutrition calculation ability as I had only calculated TPN once while taking a pre-requisite course to get into the MSND program. The Enteral Feed Calculations: Standard Formula video was very helpful, though I was a little confused at first. The video stated that though Mr. X had noted swallowing difficulties, his healthcare providers didn’t think the swallowing difficulties would be a long-term problem.(1) As a result, a short-term enteral feeding was planned and the patient would receive enteral feedings through a nasogastric tube (NG tube).(1) I learned that NG tubes are intended to be used for a few days up to a few weeks.(1) Due to the NG tube selection, the patient is most likely going to require enteral feeding for two-weeks or less.(1) I learned that overall energy needs (kcals) and protein needs are two important categories to focus on to ensure that enteral feeding will meet patient needs.(1) I was reminded that indirect calorimetry is the most accurate way to calculate energy needs, and I instantly wondered if I would have the opportunity to observe indirect calorimetry in-person. I was surprised to learn that even though indirect calorimetry is considered the “gold standard” and is one of the most accurate methods to determine energy needs, it is expensive and cumbersome, which leads practitioners to not choose this method even if it is available to them.(1) This led me to wonder how critically ill a patient must be in order for indirect calorimetry to automatically be considered. I became curious about the use of indirect calorimetry and wondered what scenario it is most likely to be used. I decided to research the literature and found an interesting study that suggested indirect calorimetry should be used in critically ill patients in the ICU as predictive energy equations have poor predictive value in all subgroups of ICU patients.(2) The study also suggested that when indirect calorimetry is not available, patients with very high and very low metabolic rates are at a significant risk of under- or over-feeding.(2) I thought this was quite interesting to learn and did not realize that if indirect calorimetry is not used in very severe ICU patients, over- and under-feeding may occur.(2) Interestingly, this study was contradicted by the Enteral Feed Calculations: Standard Formula video as the RD in the video stated that predictive equations are appropriate and can be used in critical care patients or patients on a ventilator.(1) I understand there are always nuances in science, and that one study alone does not make up the overall body of evidence – but I can’t help but wonder who is more accurate in this case. I know that working in a clinical environment, like I will during clinical rotations, should help me understand when predictive equations are and aren’t appropriate/helpful and when to use indirect calorimetry. Being in a clinical setting and observing/learning from experienced RDs will help me better understand these topics. I hope I have the opportunity to observe the indirect calorimetry process when I am in my clinical rotation. I now know that formula choice may be dependent on GI tract function, disease state, and/or volume tolerance.(1) I learned that patients who can’t handle large volumes and have fluid restrictions may need a higher concentration formula that contains all nutrients needed (meets the DRIs) but has limited free water and fluid.(1) I learned that some formulas may be created for specific disease states.(3) This reminded me of Modulen IBD, as it was created for Crohn’s Disease patients and is a big part of the Crohn’s Disease Exclusion Diet (CDED).(3) One thing that really stood out to me was that many enteral formulas contain fiber, and these fiber-containing formulas are important for patients with normal GI function to help maintain bowel health.(1) This may sound silly, but fiber in formula is not something I had thought about as the formulas I have received in the past did not contain fiber. However, I now recognize my previous formulas did not contain fiber because I suffered from altered GI function due to severe Crohn’s disease flares. What also stood out to me was ensuring the formula is appropriate to ensure the patient meets their micronutrient DRIs.(1) When I was prescribed TPN in the past, I distinctly remember two bags attached to the continuous feeding monitor (I now know I was fed with a custom 2-in-1 + lipid piggyback parenteral formula). However, I do not remember thinking about the importance of receiving micronutrients back then (this was in 2010 and I did not have an understanding of nutrition science). What I have learned from this is, if given the opportunity to work with TPN patients in a clinical setting, I will explain to them that they are receiving amino acids, dextrose and lipids (the small lipid bag is quite obvious, even to me 15 years ago!), but I will also explain that they are receiving the appropriate amount of vitamins and minerals to meet their micronutrient needs. The video Enteral Nutrition Support: Oncology Case Study was very interesting as it was a case study of a 57-year-old male with a history of head and neck cancer who was receiving radiation and was going to undergo a surgical resection in the future.(4) I wanted to watch this video for two reasons. First, our enteral worksheet presented a patient with esophageal cancer, so I wanted to further learn about how best to calculate enteral needs for cancer patients. Second, I wanted to better understand the nutritional needs that may occur in a cancer patient receiving radiation and/or chemotherapy. I learned that the first intervention this patient received was to try soft and/or pureed foods and was subsequently taught how to add protein and calories, though the patient experienced painful swallowing and difficulty eating and drinking.(4) I wondered if, in a real-world clinical situation, it would be the speech-language pathologist (SLP) who prescribed this diet modification because the patient experienced painful swallowing and difficulty eating and drinking.(4) It is my understanding that when it comes to swallowing difficulty or eating difficulty, it fell under the SLP scope of practice. I acknowledge I am not in a clinical situation and do not fully understand all nuances, however, I assess that if an esophageal cancer patient is having difficulty eating and drinking and it hurts every time he swallows, perhaps he should have been put on enteral nutrition to begin with vice trying the first intervention of soft/pureed foods.(4) For me, this is delaying much needed enteral feeding – but I fully acknowledge I do not understand the full why behind the first intervention. Perhaps the insurance company required this to occur first (kind of like step-therapy for medications), or maybe the medical clinic could not schedule the patient for a percutaneous endoscopic gastrostomy (PEG) tube in a timely manner. Either way, the patient in the case study suffered as a result. Additionally, the patient in the case study ended up presenting to the ED because he couldn’t eat or drink for ~3-4 days, which was 3-4 days after he was given the first intervention of soft/pureed foods.(4) He was then admitted for a PEG tube placement.(4) I then learned how to calculate the enteral formula needs of this patient. This video was very helpful in helping me learn not only how to choose the proper formula for the patient and how to calculate energy, protein and fluid needs, but also brought up the importance of how to initiate tube feeding. I didn’t realize that initiating tube feeding could pose such a significant risk to patients if they are malnourished or in a state of starvation.(5) I had read this in our text and also learned about it in the lecture, but a case study such as this that identified the patient as a “refeeding risk” brings it all – the lecture and text – full circle! This was very helpful for me to observe, and because I had learned about refeeding syndrome in both the text and our class lectures, and saw it again in this case study, I will not forget it. However, if I did not see refeeding syndrome in the case study, I may not have recognized a patient that may be at risk. For this, I am grateful! I will also remember that a patient at risk for refeeding syndrome also requires IV thiamine because these patients become thiamine deficient due to thiamine being a cofactor in many metabolic processes.(4) This is another example of reading about thiamine deficiency in the textbook and hearing it in the lecture, but it was the case study that brought the point full circle for me (so I won’t forget it)! The Parenteral Nutrition Calculation: Custom 2-in-1 + Lipid Piggyback video was extremely informative. The first thing that stood out to me in this video was that enteral feeding may be unsafe for an ICU patient that presents with small bowel perforation and the likelihood that the patient may require a surgical resection surgery within the next few days.(6) I was literally shocked to learn that enteral feeding may put the patient at risk for sepsis, infection, or GI tissue damage.(6) I would not have known that this could occur (again, I have never worked in a clinical setting), but this is great to know – especially since I’m going to begin my clinical rotations within the next year. This video was also a great reminder that certain medications may contain nutrients (lipids, dextrose, amino acids) and must be considered when calculating overall formula needs. In this case, the patient was on propofol. I learned that propofol is a lipid-containing sedative, and that calories are associated with the amount of propofol the patient receives.(6) I used to think that all parenteral nutrition bags were customized for the patient, but I now know that there are also standardized bags and also a variety of both custom and standardized bags.(6) This was good to know as when I was on TPN, the formula was created for my unique needs, so this led me to believe (incorrectly) that all parenteral nutrition was customized. I’m glad I learned this before starting my clinical rotation. I also didn’t realize that how the parenteral nutrition is administered is more important that simply “picking” a formula. For example, whole proteins cannot be administered directly into the bloodstream, therefore, protein is presented in the form of amino acids within the IV solution.(6) I was reminded that carbohydrates are provided as dextrose and was reminded that dextrose is 3.4 kcal/gram, which is different than the standard 4 kcal/gram for carbohydrates. It was interesting to learn that ~70% of non-protein calories can come from carbohydrates in a nondiabetic patient.(6) Equally as important, learning about the dextrose infusion rate was helpful, especially learning that the max is 5 mg/kg/min for adult patients to prevent hyperglycemia.(6) Prior to watching this video, I did not know how to calculate and account for medications that contained kcalories (in this case study it was propofol). I learned how to calculate and account for the lipids in this medication that the patient receives in a 24-hour period. It was shocking to learn that the patient was receiving 264 kcal of lipids from this medication alone! It was then I realized that if medications aren’t accounted for when calculating parenteral formula, over-feeding may occur! This was a very powerful example that I will not forget as 264 kcal is significant! It was also important to remind the patient’s care team to let the RD know when the medication (propofol in this case) is changing or if the patient is being taken off of the medication because parenteral formula needs may change as a result.(6) I understand how this can be a significant problem because if this patient was taken off of propofol, and the RD was not notified, this patient would not be receiving 264 kcal of lipids – which is significant! Something I also had not thought of prior to watching this video is that there are instances when a patient may not need as many lipids due to elevated triglycerides or another health concern.(6) In this case, a lipid piggyback may be withheld. I did not know this before and would have wondered where the patient’s lipids were if I was in a clinical environment and only saw dextrose and amino acids! I assess that if I was in a clinical setting, and had not been told this, I would have thought that the healthcare team was not doing their job as an entire macronutrient was missing! Again, this is another great example of how these videos provide an essential foundation prior to entering clinical rotations! I also now know that if a lipid piggyback is withheld for a week or more, it may be necessary to increase protein and carbohydrate provisions to ensure the patient is meeting their required energy needs to support healing.(6) I also learned that, depending on the healthcare facility, the RD may calculate electrolyte needs, but not always. I also learned that a pharmacist may instead oversee managing electrolytes. I also learned that the RD doesn’t always manage patient fluid needs, and that the ICU team may handle fluid needs instead, depending on the case.(6) The final video I watched was Parenteral Nutrition Case Study – Crohn’s Disease. The patient in this case study not only suffered from Crohn’s Disease (CD), but his CD was complicated by an enterocutaneous fistula.(7) Prior to watching this video, I had heard of fistulas but had not heard of an enterocutaneous fistula. I became curious so looked up images of an enterocutaneous fistula and was shocked and saddened by what I saw. As a Crohn’s patient, I am thankful I have not experienced this. This is another example of something I am glad I saw prior to walking into a clinical setting as I may have been quite shocked to see this for the first time inperson. I was familiar with the parenteral nutrition calculations presented in the video as this was the last video I watched. However, what stood out to me the most was that the CD patient was still losing weight despite being on parenteral nutrition for two weeks!(7) The case study patient continued to lose weight despite being on parenteral nutrition. I was not surprised that the case study patient suffered from malabsorption, but I did not know that this could persist, even if one was given parenteral nutrition. I thought parenteral nutrition rested the gut, but I then realized that the case study patient was also consuming food and drink orally. Also, the patient has a enterocutaneous fistula, which is contributing to his malabsorption.(7) My heart broke for this patient, and I was at the edge of my seat waited to see what the RDN would do next! Over the next several weeks, dextrose was increased several times and his feeds increased multiple times due to continued weight loss.(7) Over the course of several weeks, his dextrose was increased from 200 g, then up to 300 g, then up to 400 g.7 His protein increased from 100 g, then to 125 g, and again up to 150 g.7 Lastly, his lipids increased from 50 g, to 60 g, and then again up to 70 g.7 I also learned that during this time, his liver and kidney function was assessed to ensure they were not being stressed by the increase in formula (his kidney and liver function remained stable).(7) I was shocked to learn that despite feeding the patient 33 kcal/kg and 1.7 g/kg protein, he still lost weight.(7) His energy needs were then increased to 45 – 50 kcal/kg and protein was increased to 2 – 2.5 g/kg.7 With the new change, his dextrose increased from 400 g to 535 g (1819 kcal), protein increased from 150 g to 185 g (740 kcal), and his lipids increased from 60 g to 75 grams (750 kcal).(7) Thankfully, the patient finally started gaining weight with this updated TPN prescription.(7) It is an understatement to say that the increase in TPN prescription shocked me. I also felt overwhelmed and helpless for this patient as I shifted into “patient mode” and know how it feels to try to do everything “right” and continue to decline with no end in sight. (I want to insert a sad face emoji here, but that would be unprofessional – but that is exactly how I felt going through this case study). No other case study has impacted me so severely, but I assess it is because this case study is of a CD patient. I also learned what labs should be requested if a patient is on long-term PN. The labs are a baseline ferritin, total iron binding capacity, vitamin D-25(OH), RBC, folate, vitamin B12, vitamin E, vitamin A, zinc, copper, selenium, CRP, and manganese.(7) I learned to check these labs every six-months while long-term TPN is used.(7) In my assessment, six-months seems like a long time to wait to check labs for an ill patient on TPN, but again, I have a lot to learn. I also want to mention, my son watched this video with me and throughout the duration of the video he kept stating, “Poor Mr. M!” My son was very relieved to learn that the case study patient (Mr. M) healed at the conclusion of the case study. At the end of the video my son stated, “Wait – he healed? Phew – now he can start living a good life again.” To me, this underscores how much this CD case study patient suffered for several months. This also taught me that TPN is not necessarily a “quick” fix as it took this patient several months to heal, despite being on TPN. Overall, the enteral and parenteral calculation videos and case studies complimented the lecture and text reading assigned this week. They also really helped me develop an enteral and parenteral nutrition calculation foundation and, as a result, I feel more confident and prepared for my clinical rotation! References 1. Dietitians in Nutrition Support. Enteral Feed Calculations: Standard Formula. August 6, 2018. Accessed February 23, 2025. https://www.youtube.com/watch?v=tgIeGD9- _Ik&t=57s 2. Sundström Rehal M, Tatucu-Babet OA, Oosterveld T. Indirect calorimetry: should it be part of routine care or only used in specific situations?. Curr Opin Clin Nutr Metab Care. 2023;26(2):154-159. doi:10.1097/MCO.0000000000000895 3. Nestle Health Science. Modulen IBD: Explore the Benefits of Casein Protein Powder. 2024. Accessed February 23, 2025. https://www.nestlehealthscienceme.com/en/brands/modulen/modulen-ibd 4. Dietitians in Nutrition Support. Enteral Nutrition Support: Oncology Case Study. October 31, 2023. Accessed February 23, 2025. https:// https://www.youtube.com/watch?v=wck3YJLGoyk&list=PL4Q15OsBNWbS0zgbk0RJeeWhGvybnvfI&index=21 5. Nelms M, Sucher K. Nutrition Therapy and Pathophysiology. 4th ed. Cengage; 2020. 6. Dietitians in Nutrition Support. Parenteral Nutrition Calculation: Custom 2-in-1 + Lipid Piggyback. April 29, 2019. Accessed February 23, 2025. https://www.youtube.com/watch?v=bci5zL7yvWM&list=PL4Q15OsBNWb_fSYnoRsD59xGAW5ix55W&index=2 7. Dietitians in Nutrition Support. Parenteral Nutrition Case Study – Crohn’s Disease. October 24, 2024. Accessed February 23, 2025. https://www.youtube.com/watch?v=43kPg6NUWjs

Reflection

As an Inflammatory Bowel Disease (IBD) patient myself who has also been on TPN twice, it's as interesting to "be on the other side" and learn how to calculate enteral and parenteral tube feeds. I learned so much watching these videos and case studies and feel that I will be better prepared for my clinical rotation as a result. The case study that had the most profound impact on me was the Crohn's disease Parenteral Case Study (I mention this in my reflection). I learned a great deal about the complications of IBD, complications I could not even imagine. Overall, I am looking forward to starting my clinical rotation and feel that I have a good foundation on tube feeding and parenteral nutrition!

Malnutrition Awareness Week

The MAW 2023 videos really underscored that malnutrition does not discriminate in who it impacts, though some groups may suffer more than others based on demographics, weight, personal behaviors, and current living situation. The video "MAW 2023: Malnutrition in the patient with obesity – how to address the under recognition" highlighted that when looking at the epidemiology of obesity – the prevalence has increased significantly from 2000 – 2020.(1) It was shocking to learn that in 2000, obesity rates were ~15-19% in many states, or 1 in 5 people.(1) However, in 2020, many states had obesity rates of 40-45% and even as high as 50% of the population in some states being in the obese category.(1) This really underscores the fact that obesity is a chronic disease. Unfortunately, many healthcare providers and our society do not view obesity as a chronic disease and instead think obesity is a “choice.” Additionally, the obese individual is often blamed for their current weigh status. I appreciated how this video highlighted the importance of properly caring for patients with obesity and for “humanizing” them as living breathing people. As we have learned in many of our classes to date, obese people are treated unfairly and are often judged by healthcare providers. What was most shocking to me in this video is that it wasn’t until the late 1990s that NIH declared obesity as a chronic disease.(1) It was great to learn that in 2008 the obesity study joined in the initiative, and that in 2013, AMA house of delegates recognized obesity as a disease and began to focus on prevention and treatment efforts.(1) Finally, the prevention and treatment of obesity was given the same attention as hypertension, hyperlipidemia and T2DM as obesity was finally viewed as a disease that requires risk stratification and management.(1) This led me to wonder if we would have the same current obesity epidemic if prevention and treatment efforts began earlier than 2013. I assess that if “victim blaming” did not occur early on (though it still occurs now), perhaps obesity would not be associated with an estimated one-hundred-fourteen billion in annual healthcare expenditures.(1) If interventions were properly put into place in the late 1990s, when NIH formally declared obesity as a chronic disease, would we be where we are now? Would children, especially Black and Latino children, suffer from obesity at such a disproportionate rate if proper interventions were put into place in the late 1990s? I know this question is impossible to answer, but I can’t help but wonder if our obesity epidemic could have been prevented with proper education and compassion from healthcare providers and the government alike. Now, sadly, many individuals are suffering as obesity is associated with over 60 comorbidities and 13 types of cancers.(1) Additionally, all organ systems are impacted by excess weight and adiposity.(1) I was already aware that obese individuals suffer from malnutrition as we have learned this in great detail in previous classes, but this video was a sobering reminder that many in our society are suffering as a result from obesity. This video also helped give me a better understanding of obesity and malnutrition from a healthcare standpoint and what we must do from here on out to ensure obese patients are properly screened and identified for possible malnutrition. The "MAW 2023: Malnutrition and Transitions of Care Between Health Care Settings" did a fantastic job highlighting how malnutrition may occur and how it malnutrition may not be properly screened across the care continuum as patients, especially older patients, are transferred from location to location and may “fall through the cracks.” I was shocked to learn that we are an aging nation (this is not something I often think about) and that by 2035, it is expected that there will be more adults 65 and older than children

Reflection

Malnutrition is such a complicated topic as many think it is only associated with undernourishment or not eating enough food. Not meeting energy requirements and being deficient in micronutrients and macronutrients are only one aspect. Malnutrition can impact those who suffer from obesity, elderly individuals, those who suffer from chronic disease, those who suffer from cancer, and many others. In short, malnutrition does not just impact those who do not consume enough energy. It is important for healthcare providers to rely on RDNs to properly screen their patients for malnutrition, especially in long-term care facilities, the elderly population, the sick population, and everywhere in between.

Chronic Kidney Disease (CKD) Modules

Prior to working through the Chronic Kidney Disease (CKD) modules, I did not realize how interrelated diabetes, hypertension, cardiovascular disease (CVD), and CKD are. I was surprised to learn that hypertension and diabetes are the leading causes of CKD in adults, with diabetes being the most common cause.(1) Additionally, I was shocked to learn that the majority of patients with CKD die of CVD-related complications vice progressing to end stage renal disease (ESRD).(2) Prior to working through these modules, I never thought about CKD etiology and was not aware of how devastating CKD can be on the lives of those diagnosed, especially since CKD is irreversible and generally progressive.(1) The CKD modules were clear that a healthcare team working together is ideal to ensure the best patient outcomes, as this team works together to implement interventions to reduce complications and slow CKD progression. I think the collaboration of a health care team consisting of a nephrologist, dietitian, mental health professional, and social worker in addition to community support programs and patient education will give the patient the tools necessary to empower themselves, possibly slow the progression of disease, and hopefully reduce the need for kidney replacement therapy. After learning about the interdisciplinary care approach, I reflected on my own diagnosis of Crohn’s disease and know that if a similar interdisciplinary approach is taken, IBD patient outcomes may improve dramatically. These modules also helped strengthen my weakness when it comes to understanding and interpreting lab values. It was very helpful to learn that CKD is generally diagnosed if urine albumin > 30 mg/g creatinine in additional to clinical findings, and/or if eGFR < 60 mL/min/1.73m2 for more than three months. I now feel that I understand how to read and assess lab values such as eGFR and urine albumin-to-creatinine ratio (UACR). I now know that, from a nutrition lens, declining eGFR is associated with dyslipidemia, anemia and low iron stores, mineral and bone disorders, and malnutrition.(3) I also learned that additional lab values an RD must assess when reviewing the medical record of a CKD patient are BUN, electrolytes, glucose, calcium, phosphorus, albumin, serum bicarbonate, parathyroid hormone, vitamin D, hemoglobin, comprehensive metabolic panel, and complete blood count. I have made detailed notes with lab tests important for CKD, what they mean, why they are important, and what their normal ranges and abnormal ranges are for future reference and continued learning. I also intend to make flash cards so I can easily identify these lab values and what they mean. A particular lab value that stood out to me was metabolic acidosis, which was defined in the modules as a serum bicarbonate level

Reflection

Completing the CKD modules really helped give me a foundation on what CKD is, how it may be caused, what biomarkers to watch out for, and also how to provide appropriate nutrition care for a patient with CKD. This underscores the importance of the Nutrition Care Process (NCP) as each individuals CKD may be exacerbated by different issues and no two patients are identical. I now know that early intervention and timely nutrition intervention is key as CKD, T2DM, and CVD are all interrelated, so the earlier a patient receives nutrition education, the better.

Feeding Tube Placement

I used to think that the placement of a nasogastric (NG) or nasoenteric (NE) feeding tube was an easy, straightforward process that was relatively risk-free and had little to no side effects. I now know that significant patient harm and even death may occur if an NG/NE feeding tube is inserted incorrectly.(1) I was shocked to learn that malposition into the trachea or distal tracheobronchial tree could result in severe pulmonary complications.(2) These complications include tracheal perforation, pneumonia, pneumothorax, pulmonary abscess, and severe acute respiratory distress syndrome.(2) Additionally, I was shocked to learn that the blind placement technique, which is the technique used to place most of the 2 million feeding tubes in the US and UK each year, has a reported malposition overall complication rate of 10%, with 1% – 4% of this resulting in placement into the bronchial tree.(1) This means that malposition complications may occur in roughly 200,000 of the feeding tubes placed. When I reflect on this number, I realize that 200,000 is representative of actual patients who may have suffered serious consequences due to NG/NE feeding tube malposition. I am glad I now know the associated risks of NG/NE feeding tube malposition as I will do everything possible as a (future) RD to ensure that I do not cause patient harm if I am ever required to insert an NG/NE feeding tube. After watching the Compat Soft NG feeding tube placement video, I now have a greater understanding about NG feeding tube design and understand that an NG feeding tube contains five parts (stylet, ENFit connector, radiopaque tube with insertion marks, lateral openings, and rounded distal end).(3) The video also gave me a greater understanding on what must occur prior to NG tube placement, what equipment is needed for an NG tube placement, how to communicate and educate the patient prior to the procedure, how to place the patient in the Fowler’s position, how to measure the digestive tract of the patient, how to insert the NG feeding tube, how to confirm tube placement, and how to conduct daily use and maintenace.(3) It was also important to learn that NG feeding tubes are not “one size fits all” and comes in a variety of sizes, lengths, and features for both adult and pediatric patients.(3) I was also glad to learn that a patient must fast for at least 6 hours prior to NG tube placement.(3) Prior to watching this video, I was not familiar with the Fowler’s position. I find it somewhat surprising that I was not familiar with the Fowler’s position as I have been placed in this position several times while hospitalized as a Crohn’s disease (CD) patient. However, I now know that the High Fowler’s position is recommended during NG or orogastric tube placement to decrease the risk of aspiration.(4) Though outside of the RD scope of practice, it was interesting to learn that medications can also be administered via an NG tube if the medication is suitable for enteral route.(3) What stood out to me repeatedly in the video is that following and referring to “institution protocol” was mentioned in nearly every step. The video recommended following institution protocol when cleaning the patient’s nostril, lubricating the distal end of the tube, confirming proper tube placement, and follow daily use and maintenance.(3) The recommendations of following institution protocol in this video is also consistent with the ASPEN Bedside Feeding Tube Placement Competency Tool Checklist, in that multiple steps in the ASPEN checklist require things to be done according to policy.(1) The ASPEN Bedside Feeding Tube Placement Competency Tool Checklist requires confirming patient identity according to policy, placing the tube according to policy and technique for the facility, adhere by the tube placement documentation according to policy, and verify proper tube placement according to policy. Because following institution protocol or policy is such an integral part of NG/NE feeding tube placement, it highlights the importance of why a competency model for placement and verification of NG and NE feeding tubes is necessary and important.(1) Overall, I now better understand the ASPEN Bedside Feeding Tube Placement Competency Took Checklist because the Compat Soft NG feeding tube placement video provided a visual representation of NG tube placement. I am a visual learner, so I was able to remember/reflect what I had seen in the video when I was reading/reviewing the ASPEN Bedside Feeding Tube Placement Competency Took Checklist. I can say with certainty that watching the video helped me better comprehend the ASPEN Bedside Feeding Tube Placement Competency Took Checklist. It was important to learn that patients who have decreased gag reflex, decreased consciousness, or are uncooperative during the NG/NE feeding tube placement procedure are at increased risk for complications.(1) I was reminded of the dysphagia presentation we covered earlier in the semester when I learned that damaged function of deglutition or decreased consciousness may result in patients not being able to cooperate during the NG/NE feeding tube insertion as they may have reduced or absent cough reflex, which increases the chance that the NG/NE tube may be inserted into the trachea.(2) Reading this made me realize the importance of the step which requires patients that can safely swallow to drink water through a straw to facilitate tube progression when an NG/NE feeding tube is being placed.(3) This also underscores the importance of having proper training and competency prior to conducting an NG/NE feeding tube procedure. I also learned that, under the supervision of an experienced preceptor, I must successfully complete the ASPEN Bedside Feeding Tube Placement Competency Tool and must also place a minimum of 8 feeding tubes using the specified placement technique for the initial competency evaluation.(1) Prior to learning about NG/NE feeding tube placement, I assumed I would not have to place an NG/NE tube if I didn’t want to. However, I now know that RDs are often called upon to place feeding tubes and that placing an NG/NE feeding tube may be asked or required of me if I choose to work in a healthcare facility.(1,5) I also learned that direct vision-guided tube placement with a camera (Kangaroo Feeding Tube with IRIS Technology) may reduce risk as it should identify most tube-related complications because the tube position is known by operators in real-time.(6) Importantly, operators conducting the procedure would also know when the transition from the esophagus to the stomach occurs.(5) By embedding a miniature camera to the distal end of an NG feeding tube, trained clinicians are able to visually identify anatomical markers during placement, which may decrease the risk of the tube entering the respiratory tract; or, if it enters, removed pre-carina.(1,6) Amazingly, tube placement via direct visualization allows non-endoscopists (like properly trained RDs) to identify major features of the respiratory and alimentary tracts. Additionally, gastric placement via direct visualization has occurred in >90% of patients.(1,6) Importantly, direct visualization placement via camera may reduce lung trauma or infection associated with accidental placement into the respiratory tract.(6) After learning this, I am an advocate for direct visualization placement techniques. My second choice would be to conduct a real-time indirect visualization placement technique, such as an EMPD.(1) I would choose the blind placement technique as my absolute last choice.(1) While learning about NG/NE feeding tube placement, I became curious and decided to compare enteral nutrition versus parenteral nutrition recommendations for Inflammatory Bowel Disease patients. Prior to researching these recommendations, I thought parenteral nutrition was the preferred feeding method for malnourished IBD patients. I may have thought this due to my own bias, as I have received total parenteral nutrition (TPN) a few times due to Crohn’s disease (CD) flare-ups. However, what I learned shocked me. I used to think that TPN was preferred over enteral nutrition for CD patients. I now know that if oral feeding is not possible, NG/NE tube feeding is preferred, and that parenteral nutrition is reserved for patients with the most complicated disease.(7) I also now know that enteral nutrition has advantages over parenteral nutrition, which include decreased risk of infection, decreased cost, improved safety, the prevention of gut atrophy, preserving gut barrier function, and reduced length of hospital stay.(8) I also learned that enteral nutrition may reduce the rate of postoperative complication in malnourished CD patients who are undergoing surgery.(9) I am glad to now know this as it caused me to recognize my own (incorrect) bias and set it aside. References 1. Powers J, Brown B, Lyman B, et al. Development of a Competency Model for Placement and Verification of Nasogastric and Nasoenteric Feeding Tubes for Adult Hospitalized Patients. Nutr Clin Pract. 2021;36(3):517-533. doi:10.1002/ncp.10671 2. Cao W, Wang Q, Yu K. Malposition of a nasogastric feeding tube into the right pleural space of a poststroke patient. Radiol Case Rep. 2020;15(10):1988-1991. Published 2020 Aug 21. doi:10.1016/j.radcr.2020.07.082 3. Compat Enteral Access Devices. Compat Soft – How to place a NG feeding tube. YouTube; 2020. Accessed December 2, 2023. https://www.youtube.com/watch?v=cbiZOeHT_p0 4. Armstrong M, Moore RA. Anatomy, Patient Positioning. [Updated 2022 Oct 31]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2023 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK513320/# 5. ASPEN. Feeding Tube Placement Resources for Dietitians Practice Tool. January 9, 2023. Accessed December 3, 2023. https://www.nutritioncare.org/uploadedFiles/Documents/Guidelines_and_Clinical_Resources/EN_Resources/Feeding-Tube-Placement-Resources.pdf 6. Taylor SJ, Milne D, Zeino Z, Griffiths L, Clemente R, Greer-Rogers F, Brown J. Validation of image interpretation for direct vision-guided feeding tube placement. Nutr Clin Pract. 2023 Dec;38(6):1360-1367. doi: 10.1002/ncp.10997. Epub 2023 Apr 27. PMID: 37186404. 7. Bischoff SC, Escher J, Hébuterne X, et al. ESPEN practical guideline: Clinical Nutrition in inflammatory bowel disease. Clin Nutr. 2020;39(3):632-653. doi:10.1016/j.clnu.2019.11.002 8. Adeyinka A, Rouster AS, Valentine M. Enteric Feedings. [Updated 2022 Dec 26]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2023 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK532876/ 9. Lin A, Micic D. Nutrition Considerations in Inflammatory Bowel Disease. Nutr Clin Pract. 2021;36(2):298-311. doi:10.1002/ncp.10628

Reflection

After learning more about feeding tube placement, I now know why this is considered within the scope of practice for an expert level RDN. I have so much respective for those who place tube feeds. Prior to learning about feeding tube placement, I thought it was relatively easy to do and did not realize it could cause severe harm or death if inserted incorrectly. Nasogastric (NG) and nasoenteric (NE) tube placement is extremely important, and I now have a much better understanding of how much training and schooling is needed prior to placing an NG/NE tube in a patient.

Swallow Screen for Dysphagia

Prior to watching the presentation on dysphagia, I never thought about how complex the act of swallowing is and how devastating swallowing impairment can be for a patient. I used to think that the act of swallowing was a simple physiological process but now know the act of swallowing is incredibly complex and requires the activation and coordination of 50 pairs of muscles and 6 cranial nerves.(1) When learning that swallowing includes behavioral, sensory, and motor acts, I became especially curious about the sensory portion of the swallow. Prior to this lecture, I didn’t realize how important visual, auditory, or olfactory sensory stimuli are for saliva production, which allows the swallowing of foods or liquids to occur.(1) I decided to read further on the matter and found it interesting to learn that the subconscious pharyngeal swallow is triggered by sensory inputs, and this modulates the sequential motor activity of muscles that then transport the bolus through the pharynx.(2) Most interesting to me was that esophageal swallow intensity is modified by sensory input, which then triggers secondary peristalsis.(2) Learning this fascinated me and underscored that swallowing a bolus takes much more than visual, auditory, or olfactory stimuli to increases salivation, but also takes the coordination of 6 cranial nerves and 50 pairs of muscles.(1,2) Additionally, I used to think that a bolus solely resulted from the mastication of ingested food mixed with saliva, which forms a food bolus mass.(3) I now know that a bolus can also be a “volume of liquid,” and is known as a fluid bolus.(4) When learning this, I quickly realized that I once thought dysphagia was only a problem with the ingestion of food. I did not realize dysphagia also included liquids. This was very eye-opening as I once thought liquids would not pose a problem to patients who struggled with swallowing food. I also realized that I have taken swallowing for granted. I have never truly thought about swallowing physiology or thought about what would happen if an individual lost the ability to safely eat or drink.(1) I also did not think about the mental and/or social devastation that may occur when a patient experiences dysphagia. Because dysphagia can occur in all age groups and has no bias for gender or culture, it is important to realize that dysphagia may impact mental health, social aspects of life, and overall quality of life.(1) I now realize that not being able to eat or drink can throw off the family dynamic, may significantly impact mental health, and may cause feelings of social isolation as eating and drinking are important aspects of socialization in a variety of cultures.(1) Interestingly, a systematic review of 30 studies found that dysphagia is associated with depression and anxiety in patients with Parkinson’s Disease (PD), multiple sclerosis (MS), stroke, head and neck cancer, oral cancer, and tongue cancer, which underscores the impact dysphagia may have on mental health and overall quality of life.(5) I was shocked to learn that dysphagia is not a disease process but instead a symptom of an underlying disease.(1) Learning that dysphagia is a symptom that may be related to brain injury, stroke, or a progressive neurological disease like PD or MS is important to know and understand because as a future RD, I may conduct direct or indirect swallowing screenings for these patients in the future.(1) From a clinical perspective, I learned that dysphagia requires an integrative healthcare team that may consist of a medical speech-language pathologist (medSLP), registered dietitian (RD), medical doctor (MD), respiratory therapist (RT), physical therapist (PT), and occupational therapist (OT) to manage and treat a patient with dysphagia.1 It was interesting to learn that this is particularly important for patients with respiratory compromise, such as patients who have been on a ventilator or have had a recent tracheotomy, as these patients may struggle to coordinate breathing and swallowing.(1) It was shocking to learn that these patients may severely struggle as it is hard for respiratory compromised patients to stop breathing for the one-second it takes to swallow.(1) I never knew that not breathing for the one-second it takes to swallow could be so detrimental to a patient, but now know that a care plan must be in place to ensure these patients can both breath and swallow safely.(1) Knowing this knowledge will help me as a future RD as I will know that these patients may require meticulous monitoring to sustain both nutrition and hydration until they can breath and swallow safely. I used to think that dysphagia only occurred in geriatric patients, but now know that dysphagia can occur in all age groups and has no bias for gender or culture.(1) This is very important to remember as a future RD because it taught me that any patient, from pediatric to geriatric, can suffer from dysphagia and that I must always be meticulous when assessing a patient. Learning that dysphagia can present as acutely (stroke or TBI) or progressively (progressive neurological diseases or tumors) was important as this will help me as a future RD recognize when swallow screenings may be necessary. Additionally, I was thankful to learn that there are three major subtypes of dysphagia, which consist of neurogenic, mechanical/anatomical, and psychogenic.(1) Knowing and understanding the three major subtypes of dysphagia will help me, as a future RD, initiate and conduct the best possible bedside direct swallow screenings, such as The Repetitive Saliva Swallowing Test (RSST), Yale Swallow Protocol/3-ounce Water Test, and the Gugging Swallowing Screen (GUSS); or indirect swallow screenings, such as the Eating Assessment Tool (EAT-10), Swallowing Quality of Life questionnaire (SWAL-QOL), and Swallowing Disturbance Questionnaire (SDQ).1 I also know that if a patient has difficulty managing their own saliva, this is a red flag and I should consult with a MedSLP immediately for further evaluation. As a future RD, learning about the direct and indirect swallow screenings was important because MedSLPs and RDs now have the shared responsibility for the screening of dysphagia.1 Additionally, I learned that practicing these swallow screening assessments on healthy patients, such as family members or friends, will help me identify and understand what a healthy swallow feels like so that I may better identify a dysfunctional swallow when in clinical practice. I also learned that, when conducting the RSST, if less than 3 swallows are observed within 30-seconds, I should immediately refer the patient to MedSLP.1 For the Yale Swallow Protocol/3-ounce Water Test it was important for me to learn that this test is often referred to as the gold standard and is a reliable, validated screening tool for both adult and pediatric patients.1 What stood out to me the most about the Yale Swallow Protocol/3-ounce Water Test was that one of the exclusion criteria is no thin liquids due to preexisting dysphagia.(1) This particularly stood out to me because, as mentioned previously, I used to think that swallowing difficulty only pertained to food and did not pertain to liquids. However, I now know that all liquids are not created equal, and I must be diligent when assessing a patient swallowing both food and liquid. Additionally, in keeping within the RD scope of practice, it was important for me to learn that though a patient may pass the Yale Swallow Protocol/3-ounce Water Test, I should still collaborate with MedSLP to determine diet safety recommendations.(1) Importantly, I also learned that it is better to fail a patient on the Yale Swallow Protocol/3-ounce Water Test if I have a clinical intuition that something is not quite right.(1) I learned it is better to exercise caution and fail a patient than put the patient at risk for possible silent aspiration or swallowing difficulty.1 As a future RD, it was important to learn that if a patient does fail the Yale Swallow Protocol/3-ounce Water Test, they must remain NPO until the healthcare team can better understand the integrity of the patient’s airway patency and further investigate swallowing safety.(1) However, if a patient shows signs of clinical change or improvement, it was helpful to learn that the test can be re-administered in the subsequent 2-24 hours.(1) I also learned that GUSS is the true merger of the SLP and RD professions as the GUSS is one of the best, direct multidisciplinary swallow screening assessments that investigates drooling, voice, sustained attention, deglutition, and involuntary cough across semi-solids, liquids, and bolus.(1) This is due to the GUSS test being a stepwise, graded evaluation in which a patient must earn all five points from each category before moving on to the next step, and that this assessment is all or nothing.(1) This test contains 20 points maximum, and if a patient fails to reach all 20 points, a SLP must be contacted immediately for further investigation.(1) What stood out to me the most about the GUSS assessment is that semisolids are tested before liquids, which again, underscores the importance that liquids can be more dangerous than semi-solids in certain situations! This aspect was shocking to me, and I am glad I now know this. Additionally, it was also important to learn about the indirect (clinician facilitated) swallowing screenings, which consist of the Eating Assessment Tool (EAT-10), Swallowing Quality of Life questionnaire (SWAL-QOL), and Swallowing Disturbance Questionnaire (SDQ). I now know that these screenings ask a series of questions to get patient (and caregiver) perspective on swallowing and that the SDQ may help detect early dysphagia in patients with PD and other neurological disorders.(1) What stood out to me about the indirect swallowing screenings is that they empower patients by giving the patient “a voice,” as these screenings are patient guided and the patient/caregiver perspectives are obtained to identify any major concerns in addition to the impact dysphagia has on the patient’s quality of life.(1) It was terrifying to learn that silent aspiration can occur as a consequence of dysphagia.(1) I used to think that anytime foreign material entered the airway, a patient would automatically produce an overt response, such as coughing, to try and expel the material. However, I now know that silent aspiration can occur when foreign material enters the airway, and that there are often no overt signs and symptoms of aspiration.(1) However, I learned that an indicator of silent aspiration is a gurgling-sounding voice, which suggests foreign material is hovering near or has gone below the level of the vocal cords.(1) I also learned that silent aspiration may cause aspiration pneumonia, which may occur when foreign material collects in the lungs.(1) After learning about aspiration pneumonia, I became curious and found that aspiration pneumonia may be a leading cause of death in PD patients as studies suggest that aspiration pneumonia accounts for 70% of death for PD patients and may contribute to more than 10% of deaths for MS patients.(6,7) As a future RD, it was important to learn that if a patient takes too long to eat (>30 minutes), they may be expending too much energy, which may be detrimental to their health and contribute to malnourishment or dehydration.(1) Additionally, learning that patients have a range of awareness is important. For example, patients who have progressive dysphagia development due to a neurological disorder may be less aware of their swallowing safety as they have compensated for this over time by possibly eliminating food groups or have made micro-compensations throughout the years and may not realize they are at risk for dyspahagia.(1) In contrast, acute dysphagia is sudden and occurs with stroke or TBI. Learning that swallow screenings should take place within 24 hours of an acute trauma occurring was important to learn, as unscreened individuals are at a significant risk for developing pneumonia.(1) It was also important to learn that 60% to 70% of patients who have undergone radiation for head or neck cancer may present with dysphagia.(1) This is important for all RDs to understand, but especially oncology RDs, as these patients are at significant risk for malnutrition and dehydration. Prior to learning this, I would have thought that head or neck cancer patients may be malnourished or dehydrated due to radiation therapy and would not think of dysphagia as the symptom. I now know that if I work with head or neck cancer patients in the future, I must work closely with the medSLP to screen for dysphagia. Overall, learning about dysphagia has taught me so much. I now know how to identify the patient populations most at risk for dysphagia, and also know how to properly conduct direct swallow screenings and indirect swallow screenings to better identify patients who may be at risk for dysphagia.1 I now plan to practice swallow screenings on family and friends to get comfortable with the screening process so I can better identify swallowing impairment in my future patients. I also learned that complex patients must be referred to medSLP for further investigation and that dysphagia can be a multidisciplinary disorder that requires RDs, medSLPs, MDs, PTs, RTs, and OTs working together to ensure that the patient can eat and drink safely and to also work together to maintain and improve overall quality of life for the patient.(1) I am thankful I learned about dysphagia and feel that I have been given a foundation that will help me better serve my future patients. References 1. Barrera M, O’Connor-Wells B. Swallow Screen for Dysphagia: What RDs Need to Know. Accreditation Council for Education in Nutrition and Dietetics webinar. September 19, 2023. Accessed November 2, 2023. https://kings.hosted.panopto.com/Panopto/Pages/Viewer.aspx?id=759efa65-068b-4cc9-8354-b0a500b45a48&instance=Moodle 2. Steele CM, Miller AJ. Sensory input pathways and mechanisms in swallowing: a review. Dysphagia. 2010;25(4):323-333. doi:10.1007/s00455-010-9301-5 3. Heda R, Toro F, Tombazzi CR. Physiology, Pepsin. In: StatPearls. Treasure Island (FL): StatPearls Publishing; May 1, 2023. 4. Burbidge AS, Cichero JAY, Engmann J, Steele CM. "A Day in the Life of the Fluid Bolus": An Introduction to Fluid Mechanics of the Oropharyngeal Phase of Swallowing with Particular Focus on Dysphagia. Appl Rheol. 2016;26(6):10.3933/applrheol-26-64525. doi:10.3933/applrheol-26-64525 5. Khayyat YM, Abdul Wahab RA, Natto NK, et al. Impact of anxiety and depression on the swallowing process among patients with neurological disorders and head and neck neoplasia: systematic review: Egypt J Neurol Psychaitry Neurosurg. 2023;59(75). doi:10.1186/s41983-023-00674-y 6. Won JH, Byun SJ, Oh BM, Park SJ, Seo HG. Risk and mortality of aspiration pneumonia in Parkinson's disease: a nationwide database study. Sci Rep. 2021;11(1):6597. Published 2021 Mar 23. doi:10.1038/s41598-021-86011-w 7. Harding K, Zhu F, Alotaibi M, Duggan T, Tremlett H, Kingwell E. Multiple cause of death analysis in multiple sclerosis: A population-based study. Neurology. 2020;94(8):e820-e829. doi:10.1212/WNL.0000000000008907

Reflection

I learned so much about dysphagia, the importance of conducting a swallow screen, and learned that it is important that the RDN and the SLP form a strong relationship. I spoke with a nurse who was frustrated because she had taken over care of a patient who aspirated from jello because the patient was inappropriately fed after failing a swallow screen. To provide context, the patient presented to the ER with stroke-like symptoms. The patient failed a swallow screen while in the ER, however, after the patient was moved out of the ER and into their hospital room, the nurse that took over care conducting *another* swallow screen WITHOUT consulting with the SLP even though it was noted that the patient had failed the swallow screen while in the ER. The patient "passed" the second swallow screen (that was not conducted by an SLP) so the nurse fed the patient. The patient ended up aspirating on jello. I was horrified to hear this story, but also quickly recognized that the nurse failed to stay within their scope of practice. We are taught over and over again in our MSND program to always "stay with your scope of practice." This is why! I had already known the if a patent fails a swallow screen, the SLP must see the patient and we, as RDNs, must work closely with the SLP and the SLP has the final say of when and what the patient will eat. I learned so much during the swallow screen for dysphagia and will also never take the act of swallowing for granted.

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